Mutations in IL36RN/IL1F5 Are Associated with the Severe Episodic Inflammatory Skin Disease Known as Generalized Pustular Psoriasis

Onoufriadis et al., 2011

Interleukin-36–Receptor Antagonist Deficiency and Generalized Pustular Psoriasis

Marrakchi et al., 2011

IL-1F5, -F6, -F8, and -F9: A Novel IL-1 Family Signaling System That Is Active in Psoriasis and Promotes Keratinocyte Antimicrobial Peptide Expression

Johnston et al., 2011

Intricate Targeting of Immunoglobulin Somatic Hypermutation Maximizes the Efficiency of Affinity Maturation

Zheng et al., 2005

Opposing Activities of Two Novel Members of the IL-1 Ligand Family Regulate Skin Inflammation

Blumberg et al., 2007

Generation and Iterative Affinity Maturation of Antibodies in Vitro Using Hypermutating B-Cell Lines

Cumbers et al., 2002

Elevated Expression of PD‑1 on T Cells Correlates with Disease Activity in Rheumatoid Arthritis

Luu et al., 2018

Pathologically Expanded Peripheral T Helper Cell Subset Drives B Cells in Rheumatoid Arthritis

Rao et al., 2017

BTLA Signaling in Conventional and Regulatory Lymphocytes Coordinately Tempers Humoral Immunity in the Intestinal Mucosa

Stienne et al., 2022

The Inhibitory Receptor B and T Lymphocyte Attenuator Controls γδ T Cell Homeostasis and Inflammatory Responses

Bekiaris et al., 2013

Therapeutic Potential of B and T Lymphocyte Attenuator Expressed on CD8+ T Cells for Contact Hypersensitivity

Nakagomi et al., 2013

Major Differences in Inflammatory Dendritic Cells and Their Products Distinguish Atopic Dermatitis From Psoriasis